 Hi everyone, I am Dr. Sapna Mahalai, 3rd year junior resident from JMC Mumbai. My topic for presentation is role of imaging in parodic gland lesions. Parodic tumors representing 80 percent of the salivary gland tumors. Most of these are benign with pleomorphic adenoma being the most common and mucoepidermal carcinoma is the most common malignant lesion. Imaging helps in delineating the extent of the lesion and invasion of the adjacent structures. Pre-operative evaluation of the parodic gland tumors using dedicated imaging is crucial for surgical planning. The aim of this study is to localize and characterize the parodic lesions to differentiate benign from the malignant lesions to study the deeper extent of the disease to help in the early diagnosis of the lesion that are potential for malignant transformation. Role of imaging in staging of the salivary gland tumors to determine the surgical receptivity. MR imaging had been done on a patient's referred to redology department for parodic lesions. 3 Tesla MRI unit with dedicated NECOL was used for MRI. MR NEC imaging protocol included axial T1 T2 weighted images with coronal T2 star DWI sequences and post-contrast axial coronal sequences. Gadolinium-based contrast media was used. MR angiography performed with top sequences whenever needed. Agile images of the MRI sequence showed a well-defined T1 homogeneous iso-intense lesion with a T2 hyper-intense lesion represented in the superficial lobe of the lab parodic gland. The thin rim of T2 hypo-intensity representing capsule is seen. Post-contrast T1 fat-set images at the same level show your homogeneous enhancement. Based on the imaging findings, diagnosis given was the pleomorphic adenoma which came out to be positive for same. Agile images of the MRI showed a few enlarged T1 hypo-intense lesion over lesions in the superficial lobe of the lab parodic gland, appearing hypo-intense on T2 weighted images with few tiny hyper-intensistic areas within. The lesions show homogenous post-contrast enhancement with a few non-enhancing areas. True diffusion restriction were noted in a lesion. The diagnosis given was the Warthens tumor based on the multiplicity and imaging findings which came out to be positive for the same. Agile images of the MRI showed T1 hyper-intense well-defined soft tissue lesion in the superficial and deep lobe of the lab parodic gland, hyper-intense on T2 weighted images and show homogenous post-contrast enhancement with patchy areas of diffusion restriction. This was a known case of T1 disease and parodic gland involved by the same disease process. Known case of RVD since birth showed bilateral enlargement of the parodic gland with multiple solid cystic lesions and showing homogenous remenancement on a post-contrast scan. The diagnosis given was the Benign Lymphopithelial lesions. This is a known case of Type 1 neurofibromatosis in which the T2 hyper-intense large lesion is seen over the left side of the face and neck involving the lab parodic gland. Several small lesions showing central area of low intensity surrounded by T2 hyper-intense rim that is target sign is seen and the lesion appearing iso-intense on T1 weighted images and show heterogeneous peripheral post-contrast enhancement. The diagnosis given was a Plexiform Neurofibroma. A well-defined T2-star hyper-intense lobulator soft tissue lesion is seen involving the right parodic gland. Multiple plebolis are seen in the GRE sequences. Lesion appears hyposintense on T1 weighted images and early training when is seen and training into the external jugular vein. Diagnosis given was the slow flow venous mole formation depending on the early training vein. Agile and core and angiography MR sequences showed a well-defined multi-loculated cystic lesion in the subcutaneous plane of the lab vocal mucosa involving the lab parodic gland. Minimal peripheral post-contrast enhancement is seen on dynamic angiography. No evidence of any early enhancement or early training when is seen. Diagnosis given was the slow flow emphatic mole formation. Diagonal and axial sequences of the MRI showed T2 weighted isotope hyper-intense fewer well-defined lesions within the superficial lobe of the right parodic gland. Lesions show hyper-intense signal on T2-star images and homogeneous post-contrast enhancement with a true diffusion restriction. Based on the multiplicity and imaging findings the diagnosis given was the worth in steamer however it came out to be in the centric castelum and this is on a HPE. A well-defined T1-hypo-intense lesion in the deep loop of the parodic gland T2-hyper-intense lesion with peripheral post-contrast enhancement and central areas of necrosis and show peripheral areas of diffusion restriction. Diagnosis given was the carcinoma is pliomorphic adenoma which conform on pathology. A solid cystic lesion with a hypo-intense solid component on a T1 weighted images. Hypo-intense solid component on a T2 weighted images the solid component of the lesion show heterogeneous post-contrast enhancement with patchy areas of diffusion restriction on a DLWI. Diagnosis given was mycopteramidcarcinoma which came out to be positive on a histopathological examination. A solid cystic lesion in the superficial lobe of the right parodic gland with a solid component appearing hypo-intense on T1-2 weighted images. The minimal post-contrast enhancement is seen and showed true diffusion restriction. Diagnosis given was the Warthens tumor however on a histopathological examination it came out to be secreted to carcinoma. A well-defined T1 iso-intense solid cystic lesion with irregular margins is seen in the superficial lobe of the left parodic gland. Solid component appearing hypo-intense on T2 weighted images and showed heterogeneous post-contrast enhancement without true diffusion restriction. The diagnosis given was the scenic cell carcinoma which came out to be positive on a histopathological examination. Another study included 38 cases out of which plumeorphic adenoma is the most common benign lesion and mycopteramidcarcinoma with the carcinoma X plumeorphic adenoma is the most common valignant lesion. Depending on the findings in this study the most of the benign lesion showed well defined margins with a high signal intensity on a T2 weighted images and homogenous enhancement with a percent areas of necrosis and malignant lesion showed well defined margins with intermediate to low signal intensity on a T2 weighted images and heterogeneous enhancement with presence of necrosis. Plumeorphic adenoma is the most common benign parodic tumor. Lesion smaller than 2 cm showed high signal intensity on a T2 weighted images with homogenous post-contrast enhancement. Argent lesions may appear heterogeneous due to necrotic and absurd components. Barthens tumor or so known as acestadinolymphoma smoking is the main risk factor. Multiplicity and bilaterality is the future heterogeneous with variable signal intensity on a T2 weighted images and heterogeneous post-contrast enhancement. Chimora disease is a very rare chronic inflammatory disease. Interparotin lesions show homogenous enhancement with restricted diffusion due to their hypercellular nature and lower content of fibrosis. Benign lymphopithelial lesion is the mixed holistic lesion with enlargement of the parotid gland associated with the cervical lymph node enlargement. Plexiform neurofibroma is the hallmark of NF1. May show characteristic target sign. Unicentric castellament disease is also known as angiopholicular lymph node hyperplasia can be unicentric or multi-centric. Hand neck involvement is rare and extremely rare chances of salivary gland involvement. Diagnostic challenge because it mimics the other neoplasm, tissue sampling is necessary. Carcinoma, exliomeric adenoma, thought to arise from the pre-existing pleomorphic adenomas. These are generally heterogeneous on T1 weighted images. Neocorbidermal carcinomas most common malignant parotid tumor and imaging picture depend on the histological type. Acenic cell carcinoma is the most common malignant neoplasm. Almost exclusively occur in salivary gland and imaging pictures are not visible. Secretary carcinoma previously defined as a zymogen-poor variant of acenic cell carcinoma. Components are very rare, similar to the adenotic cystic carcinoma. Conclusion on MR most benign and malignant parotid lesions can be discriminated by their appearance. Obminative imaging is vital for surgical management. These are my references.