 The epithelial to mesenchymal transition, EMT, is a fundamental process by which epithelial cells acquire mesenchymal phenotypes and the ability to migrate. This process is essential for normal embryonic development and is also involved in cancer metastasis. During gastrulation, epiblast cells ingress from the primitive streak to form mesoderm. The ingression of these cells is dynamic and involves a stereotypical sequence of cell behaviors, including the asynchronous ingression of epiblast cells and the concomitant shrinking of their apical surface. This shrinking is driven by the coordinated contraction of actomyosin networks and crumbs two complexes, which are important regulators of this process. These findings provide new insights into how epiblast cells ingress and suggest that crumbs two may be a key regulator of the EMT process. This article was authored by Alexandra Franco, Catherine V. Anderson and Anna Caterina had gentanacus.