 Hello everyone, my name is Dr. Ramadhan Amnath Kutty, I am a third-generation resident in the Department of Relative Diagnosis at Dr. Ullas Medical College and Hospital Jharka of Maharashtra. My topic for presentation is to evaluate the role of magnetic resonance imaging in AVS-Clinic Races of Immoral Head. Introduction to AVS-Clinic Races or Austinic Races is defined as cellular death of bone due to decreased blood supply which can be required due to causes. The interaction first starts as the blood supply to the bone is interrupted. Once the interaction sets in, there is central necrosis which is surrounded by peripheral ischemic zone. Beyond this zone lies the normal viral bone irritation. Various factors causing AVS-Clinic Races have been identified which interrupts the blood supply, the affected bone and part of it. AVS-Clinic Races of Immoral Head is most common among the affected sites of AVN presumably because of its precarious blood supply and high loading during standing. These factors trauma, alcohol abuse, corticosteroids, magnetitis, sickle cell disease, infiltrative diseases, radiation. Aims an objective to study the stage of presentation of AVS-Clinic Races of Immoral Head in magnetic resonance imaging. Analyze the role of magnetic resonance imaging in patients with AVS-Clinic Races of Immoral Head. Staging of AVS-Clinic Races of Immoral Head as for wicked and early classification. Models and methods. Descriptive study of medications where clinically diagnosed with AVS-Clinic Races of Immoral Head and referred for MRI of the both hip joints to the department of radio diagnosis. These patients were subjected to screening over 1.5 TESLA CMS machine. T1, T2, STER and gradient inconsistencies were applied in multiple lanes. Inclusion criteria. Clinically suspected AVN of Immoral Head. Patients with AVN involved concern for the study. Patients with STF or trauma and suspected to have AVN. Exclusion criteria. Patients who refused to participate. Patients with contraindication to MRI. Like cardiac pacemakers that are magnetic implanted close to the area. As well as we get an early stage one. There is a number of edema. As we can see in this T1 coronal and T2 axial image. In stage two there are geographic areas of altered signal intensity and double line sign. In left side model here we can see double line sign in this T1 and T2 coronal images. In right side model here we can see the geographic areas of altered signal intensity. With cortical collapse. Where it has increased to stage three. In stage three there is a crescent sign with cortical collapse. As seen in this coronal cluster and data sequence. In both of the temporal heads. In stage four all the signs are stage three. With the secondary degenerative changes. In surrounding acetabulum. And as seen in this T1 and T2 coronal images. Staging of AVN. As well as we get an early classification. Patient presented. In stage one. In stage two. 25 patients. In stage three. 13. In stage four. 10. So most of the patient presented. In stage two and three. And these were double line signs. In 34 patients. In 28 joint division. In 25. In 16. In 23. So double line sign was the most common finding. Gender distribution. About 70,000 information per male. 26,000 information per female. Laterally talked about AVN. In the right side. In the cases where bilateral discussion. There was a male patient. AVN was bilateral in most of the cases. With alcohol consumption being the most common. Causative factor. Patient presented in a stage two. As well as we get an early classification. Double line sign was the most common. In the right side. Studied cases. As well as the necrosis. That is the common. In the right side. In stability at the hip joint. During early stages of AVN. Patient may remain asymptomatic. Using challenge for a clinician. Making MR imaging. In distance for early diagnosis. And management. Staging as per we get an early classification. Stage zero. Plain radiograph MRI are normal. With no significant clinical symptoms. In stage one. Radiographs may be normal. Or a minor osteopenia may be seen. MRI bandings are bone marrow edema. Clinical patients present with pain. Typically at groin. In stage two. Plain radiograph shown mixed osteopenia. With or without sclerosis. Without any. Subcontinuosency. MRI show. Geographical defect. Clinically. Patient present with pain. And stiffness. In stage three. There is a crescent sign. With eventual cortical collapse. MRI is the same as. Plain radiograph. Clinically. There is a pain. And stiffness. With or without. Radiation. Knee. And patient may have a limping. In stage four. Plain radiograph. Shows end stage disease. With evidence of secondary degenerative changes. In surrounding bones. MRI. Same as a radiograph. Clinically. Patient present with pain. And limb. Other classifications. Michiel classification. Steinberg classification. Arco classification. Conclusion. MRI is indispensable for early detection. And accurate staging of avian of femoral head. It can define size. Sight of the legion. Which is helpful in estimating stage of the disease. Thus over the years. MRI has become the most sensitive and specific imaging modality. Or evaluation of avian of femoral head. These are my references.