 I'm going to present a case series on intramarticular hemorrhage. I'm from the US Indian Department of Radiology from S.T. Numbangwai. Astroliter intramarticular hemorrhage is rare in adults and seldom described. After ruling out the history of trauma in US, etiologies remain possible. For most frequently found underlying reasons are atrovinous malformations and organisms. But other vascular causes we need to rule out are cavernous malformations and myemorrhages. Intramarticular tumors can also present with IVH. Atrial hypertension, anticoagulant use, coagulopathy, sensorine toxin, substance absorption with IVH. Finally, the IVHs can be idiopathic. And the necessary investigations, CT angiography, MRI angiography needed to be sought out for finding the cause. This is the aim of the pictorial assay of astroliter with the etiology of intramarticular hemorrhage. Vascular pathologies, AVM, aneurysm, cavernous malformations, voiomboid disease, duolefistry, and vascular disease of the avascular pathologies tumor, intramarticular tumor, producing factors like atrial hypertension, coagulopathy, anticoagulation symptoms, trauma, surgery, and the circulation of subarachneuritis. In case of bilateral intramarticular hemorrhage in the lateral matricle, the cause was found to be moiomboid disease. There is a narrowing of the internal carotid artery with complete occlusion of the supra-clinid portion of the internal carotid artery and multiple collaterals are seen. So, multiple collaterals are noted around the circle of the village supplying the anterior circulation from branches of posterior cerebral arteries. Likely suggested of moiomboid disease and there was complete occlusion of bilateral supra-clinid segment with narrowing of bilateral cervical and intravenial portions. In the case of moiomboid disease, which presented with isolated bilateral intramarticular hemorrhage, in case of intramarticular hemorrhage, fourth ventricle and some subtle subarachneuritis hemorrhage, in this case it was found to have thrombosis of the internal carotid artery, complete thrombosis of extra-clinial internal intra-clinic internal carotid artery. In this case of the bilateral intramarticular hemorrhage with subarachneuritis hemorrhage and the cause was acohomortitism, which is present, measuring 8.8 into 7.2 into 9.6 m. Next, in case of quantifier old female with sudden onset and responsiveness, isolated ventramarticular hemorrhage, third ventricle and lateral ventricle, third ventricle and fourth ventricle and the cause was found to be the multiple collateral rising from the posterior cerebral arteries. There is an idus here present and the draining vein into the internal cerebral veins. So, this is the arterovineal smart formation. Fifth-year female with cerebral vascular accident, intra-ventral hemorrhage noted in the left lateral ventricle and there was found to have ABM in the spleenium of corpus callosum, posterior to the spleenium bilateral middle element with the cyclone and resumon dilatation, nidus measuring 11 into 13 to 10 mm, feeding artery from right posterior cerebral artery, F3 here from the right posterior cerebral artery and the draining veins into bilateral internal cerebral veins and middle ventricular veins, which in turn was drained in the spleen-draining-performed-prepared internal straight sinus, straight venous sinus, again into supracellatoid. So, this case of complex ABM formation with aneurysm. So, ABM formation can lead to hemorrhage restricted to the ventricular system and have been identified as causus of intramarticular hemorrhage. Another case of 35-year-old with headache, blood hemorrhage, right lateral ventricular hemorrhage and pitifiable angiogram was within the normal limit. So, this is one of the cause, where we don't know. Intramarticular bleed, Z-ary homing, there was on T2 major, there was a tumor, large loblated myelianus interventory lesion was present with normal resistance with tumor bleed and intramarticular extension was also present. So, conclusion is a variety of pathologies are responsible for isolated IVs. So, we have to do CT angiography, MR angiography and catheter angiography is must. It provides complement information for the concurrent evaluation and a potential treatment access. Thank you.