 Hello, Namaste. I'm Dr. Harthik Raihani, second year resident, B.J. Medical College, U.S. Hospital, Ahmedabad. Today, I am presenting a paper on the role of MRI in congenital CNS malformation. For this, my guiding role was Dr. N. A. Patel. He is an assistant professor at CM Medical College. The aim of my paper was to characterize various congenital CNS malformation on MRI. For this, we have studied 20 patients who have come to our department from January 2020 to February 2020, and we have performed all of these scan on the 1.5 Tesla Philips MR scanner. In today's discussion, I have included nine cases from that 20 patients with a pictorial review. First case was of occipital and cephalosil. Here is the 15 days old female child who was presented with cystic swelling over occipital region. On T2 weighted MRI images, we have seen that there is occipital region and cephalosil. And there was also a non-communicating hydrocapillus with partial agenesis of corpus callosal. The second case was of corpus callosal agenesis. Here, there is a one year old female child presented with hypotonia, scissor and delayed milestones. On T1 weighted societal images, there is a complete absence of corpus callosal. The singular sulcus is absent. The medial hemispheric sulci leads the third ventricle in radial crescent. In procedural section, through the lateral ventricle, there is a dilatation of the atrium and occipitolone suggestive of corpus callus. On T2 weighted coronal images, there is a wide divergence of frontal wounds and elevation of the third ventricle with medial indentation on the frontal wound represented by corpus bundle. On XL T1 weighted images, the lateral ventricle are parallel to each other and do not come in contact with each other as they normally should. The third case was of Arnold Kiari malformation type 2. Here, the one month old male child presented with back swelling in the lumbosecral lesion. On MRI, there is a lumbosecral meningo myelosil. In posterior fossa, there is a vermin descent through the foramen magnum medullary kinking, tectal beaking, hypoplastic left cerebral hemisphere, dilated both lateral and third ventricle and plus slit like fourth ventricle. So, it was a case of Arnold Kiari malformation type 2. The fourth case was of Dendi vocal malformation. Here, the nine month old male child presented with dilate development, irritability and progressive enlargement of the skull. On MR examination, there is evidence of large posterior fossa cysts communicating with enlarged fourth ventricle and there is an absent cerebral vermin. The fifth case was of the scisensiphalin. Here, the seven days old female child presented with atectable seizures. On MR examination, there is evidence of large full thickness cleft in the left cerebral hemisphere communicating with ventricle. The cleft is lined by the grade matter. The sixth case was of sub-ependymal heterotopia. Here, the four year old female child presented with refractory scissor disorder, however, development was normal. On MR examination, there is a multiple grade matter iso intense nodules lining the lateral ventricle. The seventh case was of Lombar holopresensibility. Here, the five month old female child presented with dilate milestone, intact able scissor. On MR examination, there is evidence of partial fusion of the frontal lobe and incomplete corpus callosum. Both lateral ventricle as well as the deep grade matter nuclei are known. The case eight was of the equidectal stenosis. Here, the one year old female child presented with increasing head size, scissor, vomiting and sunset phenomena. On MR examination, there is an obstructive hydrocephalus with marked enlargement of the lateral ventricle, third ventricle and however, the fourth ventricle is not dilated. The ninth case was of Vainov Galen malformation, which is the misnomer. It was the case of proxen... In this case, the two month old child presented with palpable pulsation over fontanel and scissor. On MR examination, there is evidence of dilated venous sac in the region of Vainov Galen with persistent mesentrophilic vein and dilated pulse and sinus bending into the proclure hetrophile with dilation of posterior part of supersonic lateral sinus in both transverse sinuses. Multiple vascular channels noted arising from the posterior circulation with communication with above mentioned venous malformation, which we see on this NGO reconstruction. And here is the dilated vein. In the discussion, the first case of cephalocephaly is the brain or CSF filled dural herniation with the skull defect. It does not show enhancement unless vessel or dural venous sinuses herniated through it. So, we mostly perform MRI with Vinogram to see the differentiation. Corpus callosal disinase is the most common anomaly seen with other sinus malformation. If isolated finding it is more common in males, patients are near normal up to three years. And not clearly malformation are the group of disorders created by increasing degree of hind-brain disinase. In type 1 cerebral tonsil herniate, in type 1 cerebral tonsil herniated through poramina magnum. In type 2 inferior vertebra means 4th ventricle medulla herniated through the poramina magnum with small posterior fossa. In type 3 occipital encephalosil associated with hind-brain malformation. And herniation. Dendi-vocal malformation is the cystic dilatation of 4th ventricle with enlarged posterior fossa and superiorly rotated vermin and remnant. Cisansiphaly is the congenital brain malformation characterized by the cleft extending from the file surface of cerebral medulla to the ventricle and lined by polymichrogyne cortex. Hydro-topic grémical occur because of arrested neuron along the migration path from peri-ventricle germinal zone to the cortex. Equiductal stenosis is the focal reduction in the diameter of equiductal sylvia responsible for 20% of congenital hydrocephalus. Lobar holemplation is the fusion of frontal lobe with normal formed interspheric freezer and lateral ventricles and incomplete absence of anterior corpus callosum. Median personal catholic of Markovski malformation or also known as venop gallium malformation is the base diagnostic clue is the dilated vasculogenus feeding into the large midline venous pouch. From the study we have concluded that MRI is the best imaging modality to diagnose and characterize the lesion as well as for screening of the rest of the CNS to look for other CNS abnormality. Appropriate clinical as well as radiological examination will lead to definitive diagnosis of various congenital CNS malformation with surgical or medical manager of that cases. Thank you.