 Good morning, everyone. I'm presenting a paper on disseminated cystocircuses, a rare case report. Introduction, human cystocircuses is an important cause of epilepsy and neurological morbidity in developing countries. The cystocircuses is caused by the cystocircus cellulose, iloroastase of tapeworm, denia sodium. The commonest form is neurocystocircuses. Cis stalkers, especially in the strident muscles, subcutaneous tissue and the nervous system and the eye. The cystocircuses become symptomatic almost exclusively in the nervous system and the eye. Case presentation, the 35-year-old male presented with one episode of conversion and also sensory. History of poor intake occasionally. Then we did a city brain plane, which shows multiple subcentrometric lesions with eccentric calcified foci, and are noted scattered diffusely in bilateral cerebrals, cerebellar hemispheres, including DC region and ventricular system recti muscles, the eyes, skull, all-paravetable neck muscles, and posterior one-third of the tongue. Here we can see the cyst scoliosis in cerebellar hemispheres. Here we can see the cyst in ventricular system. Here we can see the cyst in recti muscles, which appears here in cerebellar region. We can see the cyst in paravetable neck muscles and posterior one-third of the tongue. We can clearly see the cyst scoliosis in recti muscles in paravetable neck regions. Then we did the MRI brain plane plus contrast of this patient and which shows the hyper intense or iso intense lesions on flare here in ventricular system or in skull. You can see many cysts in both the hemispheres of cerebral and cerebellar hemispheres in recti muscles and in skull region. Here we can see the multiple cysts in recti muscles on the both eye and posterior one-third of the tongue muscles and recti and paravetable muscles. On MRI it shows multiple thick wall cystic lesions noted in both cerebellar and cerebellar hemispheres, including the ganglothalamic and bilateral ventricles, which appear hyper intense on T2 weighted images and hyper intense on T1 and flare shows restriction on DWI no blooming on CRE. A lesion shows peripheral ring enhancement on post-contrast study with central non-lensing component. The similar morphological lesions noted in skull region both orbit same for a temporal fossa and tongue neck muscles. Diagnosis may be disseminated in cysts to circuses. In case discussion, the neurocystere circus is most common parasitic infection of the brain and the leading cause of epilepsy in the developing world. The major forms of neurocystere circuses are parenchymal, ventricular, subbreconate, spinal, orbital, ventricular, basal system locations are considered to be malignant form as the mortality rate is high in the hydrocephalus if hydrocephalus is present. The extensive involvement of brain, eyes, muscles, spinal cord and subcutaneous tissue is rare. The extracellular cysts are a slow-growing tumor or a nodal with focal inflammation. In our patient, we had cyst in rectus nodules but did not have any ocular complaints. The cyst in muscles may manifest as a muscular pain, weakness or pseudo-hypertrophy. The subcutaneous cysts to circuses is frequently asymptomatic but may manifest as in polypapal nodules. MRI is considered the best neuroimaging tool for a logical diagnosis of neurocystere circuses with advantages of being able to differentiate the stages of parasite. The stages of neurocystere circuses, there are four stages. In the first, the pathological stages, developmental stages, the circular stage, there is no surrounding edema or there is no enhancement. The MRI shows that the cyst is isointestinal to CSF and the scolex is discreet and nodular hyperintense which appears as a target region. The enhancement is typically absent. In colloidal stage, the cyst is hyperdense to CSF on CCT and M1 straight ring enhancing capsule on CCT. MRI shows that the cyst is hyperintense to CSF on T1 weighted images and the scolex appears hyperintense on flare with surrounding edema. And the enhancement of the cyst scole is typical. In granular stage, there will be mild residual edema with enhancing nodule. And nodular or feint ring like enhancement is typical. In nodular or calcified stage, the nodule without surrounding edema or calcified lesions appears as hyperintense on T1 and T2 weighted images and booming on GRE. The management of neurocystere circuses includes antiparasitic drugs like alvendazol, tizicontal, antisesia prophylaxis and symptomatic medications. Thank you.