 Hello, everyone, myself, Dr. Vinay at the PC resident department of radio diagnosis, ABVMS and Dr. RML hospital, New Delhi. Title of my paper is role of multi-detector computer tomography in evaluation of congenital cyanotic heart disease. Introduction, congenital heart disease are considered as the most common congenital birth defects comprising around 1% of all livers. CSDs have two types, a, cyanotic and cyanotic, depending on the presence of sinosis and physical examination. In cyanotic CSD, systemic venous blood bypasses the pulmonary circulation and gets shunted into the left of the heart. MD CT can show the morphology of extra cardiac vasculature including the coronaries, pulmonary arteries, aorta and pulmonary or systemic veins. And it delineates the vessel walls and also displays the airway medicinal abnormalities and pulmonary parenchyma. Aim to find out the role of MD CT in diagnosis of congenital cyanotic heart disease. Materials and method, venue of studies, the study conducted in our department, study type is cross-sectional observational study. Study duration is 1st January 2022, 1st July 2021, sample size is 60 cases, inclusion criteria. Patients of pediatric age group who have been clinically suspected to have cyanotic congenital heart disease were sent to our department and were included in the study. Methodology, MD CT on 128 slides seamen somatom was done in all patients suspected to have cyanotic congenital heart disease, based on clinically and preliminary examinations. Results, a total of 60 patients of cyanotic congenital heart disease were included in the study, out of which 20 patients were of TOF, 6 patients were of truncus arteriosus, 10 patients were of tricuspid atrazia, 10 patients were of TGA and 14 patients were of TAPVC. Now cases, first is TOF, Tetralogy of Phthalate. Here we can see a sub aortic ventricular septal defect and with more than 50% of overriding of aorta is also seen and there is a severe infundibular and supravalular pulmonary stenosis is seen. Here right atrium is dilated and right ventricular is hypertrophic. We can also see small map cause that is major aortic pulmonary collateral arteries. Now second case is TGA, transposition of great arteries. Here cytosine versus is present because the stomach and the spleen on the right side and liver on the left side. And here morphological right atrium is on the left side and is joining the left ventricle and the morphological left atrium is present on the right side and is joining the right ventricle. And aorta is seen arising from the right ventricle and the pulmonary artery is seen arising from the left ventricle. So this is a transposition of great arteries. So by one product, this is congenital corrected TGA with cytosine versus mesocardia and atrio ventricular and ventricular arterial disconcordance. Now third case is tricuspid atria. In this the morphological right atrium is dilated and has no direct communication with the right ventricle. The right ventricular is hypoplastic and is associated with tricuspid atria. Here we can also see Ostium primum type of ESD with dilatation of the right atrium and the left ventricle. There is a aberrant right subclavian artery seen arising from the posterior aspect of arch of aorta and going posterior to esophagus. So by one product it is cytosolitis, levocardia, AV concordance and D loop ventricular topology. Now fourth case is truncus arteriosus bit off. Here we can see a sub aortic peri-membranous VSD is seen with more than 50% overriding of aorta. And pulmonary valve is atria seen in this case and the left pulmonary artery is seen arising from the little part of the truncus and right pulmonary artery is hypoplastic. And so right lung is supplied by the collectors found by the bronchial artery. So this is the case of truncus arteriosus by one block system it comes in A3 classification. Fifth case is supracardic TAPVC. In this the right upper lobe and right lower lobe veins joined to form a common channel superior to the left atrium. And the common channel is joined by another trunk found by the left upper and left lower lobe pulmonary veins and this combined to form a vertical vein. So vertical vein is seen to ascend anterior and to the left of the pulmonary artery along the medicinal border to drain into the left brachiocephalic vein. In this case the left brachiocephalic vein, the SVC, the right atrium and the right ventricle are dilated. And large osteumatic secondum ASD is also seen by one block system it is SDS and AVVA concordance. Next case is cardiac TAPVC. In this the SVC and IVC are draining into the right atrium and the pulmonary veins are seen to drain into the coronary sinus and then into the right atrium. And so coronary sinus and right atrium are dilated and we can also see a small muscular type of VSD is present. And by one block it is cytosolitis and levocardia AVVA concordance and D lobe ventricular topology. Next is infracardic TAPVC. In this the pulmonary veins are converging behind the left atrium and they are forming a common descending vertical vein which is draining into the left pulmonary vein, sorry, portal vein. And the SVC and IVC are draining into the right atrium and the ground glass opacities are seen in the bilateral lung fields due to the pulmonary edema along with the bilateral pleural effusion. And cytosolitis and levocardia and AVVA concordance. Now discussion, congenital heart disease are considered as the most common congenital blood defects comprising around 1% of all live births. They have two types, asynotic and cyanotic. Among cyanotic, we have TGA transposition of great arteries. In this the complete transposition of great arteries is occur because and it occurs in the combination of atrioventricular concordance and ventricular arterial disconcordance. That is ascending aorta arises from the right ventricle and pulmonary artery arises from left ventricle. And in congenitally corrected TGA is a condition in which the both AV and VA disconcordance is present. Next is tricuspid atrasia. In this the morphological right atrium has no direct communication with the right ventricle and there are two types. Most common is when the right atrioventricular connection is absent and aerial soft tissue occupies the gap in rare type and atritic tricuspid valve is present. Third is trough tetralgia phalate. It consists of sub pulmonary infundibular stenosis, overriding of aorta, ventricular septal defect and right ventricular hypertrophy. Fourth is truncus arteriosus. In this there is a single arterial trunk arises from the ventricles and through which a single arterial valve supply both the systemic pulmonary and coronary arterial circulation instead of separation of aorta and pulmonary artery. Fifth is TAPVC, total anomalous pulmonary venous connection. In this it occurs when the pulmonary veins fail to drain into the left atrium normally and form an aberrant connection with some other cardiovascular structures. There are basically three types supracardic TAPVC in which pulmonary veins drain into vertical vein with joint left inominate vein and further joint right inominate vein to form SVC and then drain into the right atrium. Cardic TAPVC in which the pulmonary veins are drained into the coronary sinus and into the right atrium. Infracardic TAPVC in which the pulmonary veins converge to form a vertical vein which descends to enter into the portal vein and it is always obstructive type due to vascular resistance of the liver. In conclusion, MDCT proved to be an important modality for decision making in patients with congenitally cyanotic heart disease. References. Thank you.