 to all my C.S. Dr. in Pina, you may refer to my department I am a medical college in the M.P. My paper is talking to a spectrometer to find out is there is no Texas engine. But we will start with the introduction. Basically, flexes that include C.F.I.P.T. and NARU are also there. The work of the course of regular Texas injuries is taught by the M.P.T. and NARU. Clinical investigation of peripheral nerve injuries to improve nerve conduction study and electron eye graft. Imaging study is limited to extruding focal mass region and external compression to the nerve. MRI can identify changes in peripheral nerve and secondary neurogenic alterations in skeletal muscle due to excellence of tissue decay. MRI is helpful in diagnosis and localizing size of injury on this flow nervous system. Contrast study is usually done in patient with tumor or mass region and gadolin is known administered in patient with traumatic break-in flexor. These are the common root of the break-in flexor injury including burp injury, auto vehicle accident, or fall or high-prone disease. Then classification of the traumatic break-in flexor fatigue. We can classify according to real classification, sedent classification, sedent classification, sedent classification, break-in flexor injury is divided from three types for neuro, pulmonary flexia, axonotomiasis and neurotomiasis. In Sunderland classification 6 degrees are there. So first we will start with neuro flexia. It includes Sunderland classification first degree injury. It includes impaired nerve conduction with intact endangolium ferric helium and endangolium layer. And usually recovery is slightly related by glidesol lipid. And usually we manage tendermitis. In axonotomiasis Sunderland classification 2nd, 3rd and 4th degree re-routed. In 2nd degree, axonal disruption with intact endangolium ferric helium and endangolium methods. In 3rd degree, axonal and endangolium disruptions with intact endangolium ferric helium and endangolium layer. And regeneration is usually handled by mild fibrosis and recovery is likely with mild endangolium. And management is subjective. In 4th degree, axonal, endangolium and perineural disruption with intact endangolium layer. Regeneration is handled by fibrosis. In 5th and 6th degree of Sunderland classification is included. In 5th degree, complete now description is there. And in 6th degree, any combination of fibrosis will be included. In usually both cases there is no chance of spontaneous recovery. And surgery is usually considered. So this image we can see various types of the injuries. Last one is neurobrexia of secondary axonal premises. Then second one is cost ganglionic Tf and third one is 3 ganglionic Tf. Then we will discuss which now are affected in which type of lesion. In supra-clavicular lesion, roots of C-hydrate even now are affected usually. And other now are phoenic now, thoracic now, transglock upper, middle and lower now are affected in supra-clavicular now are other now are affected in supra-clavicular lesion. In clavicular lesion usually division of upper, middle and lower are affected. In supra-clavicular lesion lateral and middle and posterior core of the now are usually affected. Then first we will see 3 ganglionic Tf. Foment phoenics are edema of the spinal cord, hemorrhaging now on spinalisic band and pseudo meningocene. Ideally imaging of the suspected free ganglionic injuries were performed at least 3-4 weeks after the injury as this allowed the results of the acute edema and subrachlone m-ray and formation of a pseudocrine. So this is the first case patient presented with symptom of right side partial ptosis and myosin, likely a hormonal syndrome. There was a history of the m-ray was performed and it shows a pseudo meningocene formation in C7, C8 and T1 now roots at the level of neural foramina, suggesting free ganglionic now root was an injury. Second case was a 6 year old child generated with weakness and left hands since birth with poor reflex, sensory loss, mildishness, joint muscle weakness and atrope. There was a history of assisted delivery of healthy care. M.R.1 was found a well-defined dumbbell sacred cystic lesion in left C7 to T1 now roots and left T1 to T1 T1 and T2 neural foramina which is extra direct and exchange just beyond the neural foramina. The right C8 and T1 now roots are now visualized suggestive pseudo meningocene and a free ganglionic brachial plexus into it. Then we will see those ganglionic