 myself, Dr. Vivek Yadav, J.R.3, Department of Registrarialology. My topic for oral presentation is the evaluation of focal apathetic lesion by triple-phase CT scan. My co-author are Dr. Mukta Muttal and Dr. Sachin Agarwal. Center of affiliation is Satyapati Sivaji Sivarthi Hospital, Mayor of Uttar Pradesh. Introduction with the wide-spread of cross-sectional imaging, a growth in rate of incidentally effective focal liver lesion has been observed. A reliable detection and characterization of focal liver lesion is critical for optimal patient management. The majority of focal liver lesion arising in the non-serotic liver are benign. Even in patient with a known extra apathetic malignancy, cisthemeanoma focal nodular hyperplasia and hepatosolidoma are most commonly encountered benign lesion. Most commonly encountered malignant lesion in non-serotic liver are metastasis. Hepatosolidoma and intri-epathetic palenjo-carcinoma are mainly in setting up chronic liver disease and represent most common primary liver malignancies. Benign lesion is hepathetic hemangioma. It is the most common benign lesion diagnosed at any age. The majority of lesion occurs at between the 30 to 50 years of age. In adults, hemangioma occurs most frequently in the female with a ratio of approximately 3 to 1. Leisure more often occurs after solitary, but multiple hemangioma may also be present. Focal nodular hyperplasia is benign liver lesion that is composed of a proliferation of hyperplastic hepatocytes surrounding a central, sterile scar. Typically, focal nodular hyperplasia is a solitary lesion that is more commonly occurred in the women. Hepatosolidoma and uncommon solid benign liver lesion that develop in an otherwise normal-appearing liver. Typically, these are the solitary and are found in young female in a system with the use of estrogen-containing medication. In addition, patients with glycogen and estrogen disease and metabolic syndrome are at high risk for developing epatosolidoma. Regenerative nodules develop in response to liver injury and are compromises of a proliferation of hepatocytes and surrounding stroma. They are typically seen in the setting of cirrhosis. Benign lesion hepatosolidoma is the primary tumor of the liver that develop in the setting of the chronic liver disease, particularly in patients with cirrhosis of any gauze or chronic hepatitis B or hepatitis C, who are the same person. Polyangiocosaloma is a regis from the epithelial cells of intrapartic duct and extrapartic bile duct and may be found incidentally during the early stage of the disease. In the United States and Europe, the main risk factor are the primary sclerosis, polyangiocytes, and fibro-policistic liver disease, while hepatitis is commonly associated with Polyangiocosaloma in Asia. Metastasis, the liver is a common site for metastasis from solid tumor and patient with a history of malignancy are at high risk for metastatic disease. It aims an adjective to evaluate focal hepatic lesion across all age group by triple phase CT to assess the imaging characteristics of triple phase CT in different CTs benign from malignant focal hepatic lesion to correlate triple phase CT imaging finding with histopathological finding wherever possible. Materials and metals, sample size minimum of 50% is from October 2020 to September 2020. Inclusion criteria, all focal hepatic lesions diagnosed on ultrasonic therapy or previous imaging, all cases of chronic hepatic disease. Inclusion criteria are the pregnancy, derailed function test, patient who do not give function, patient with finding of simple cyst on ultrasonic therapy. Methodological clinical history will be recorded, check renal function test, after taking informed patient from patient, the quadriple phase CT scan will be done by radiology department. And with this MVCT examination will be done on Philips in humanity 128 slides, imaging and diagnosis finding will be made as per department protocol. Histopathological lesion will be done wherever possible. The results will be tabulated, analyzed and selected to statistical analysis wherever possible. This is the ICB protocol, first is the pre-contas, second phase is the atrial phase of pentasynarthritis, it is a empirical delay after 35 seconds or a bolus rigor. Hepatic artery and putter vein enhance but not hepatic vein, if no putter vein enhance is too early, if see hepatic vein is too late. To detect hypervisceral lesion, putter vein is phase of pentasynarthritis, empiric delays of 70 to 80 seconds, putter vein, hepatic vein and hepatic vein enhance. This test to enhance the perinchaima detected hypervisceral lesion and evaluate for lesion washout or casual enhancement. The equilibrium is empirical delay of 3 to 5 minutes, this test phase to evaluate perinchaima's lesion washout, casual enhancement and delay enhancement. Adrenal CT is completed to the pelvic level, if hemangema is suspected additional delays image to assess for feeling enough hemangema. The result a total of 50% were included in the study out of this, out of this 26% were male and 24% were female. Distribution of benign lesion is 6% and benign lesion is 44%. Out of 6% of benign lesion, 5% of hemangema, 4 are typical and 1% of benign lesion. 31% of primary valency and 69% of secondary. In metastasic lesion patient, 53% came from primary GB mass and more than 70% are hypervascular. Out of 11% of hepatocerebral plasma, approximately 90% other than serotic patient and 55% associated with the hepatic C virus and approximately 70% associated with the increased alpha vitro protein. In the case, in plain images, there is a hypodense lesion seen in the in the most segment of both the upper lipid. On arterial face is so peripheral nodal enhancement. In the last phase, there is a progressive central care feeling. This is the case size 2 of the hemangema. This is a known case of hepatitis B virus with raised alpha vitro protein. We can see on plain images, there is a nodal margin of liver with cordate and left lobe of liver in the arch associated with the SITs. There is a hypodense lesion seen in the right lobe which enhance heterogeneous lesion on arterial face and washout on bilage face. This is the case of hepatocelular carcinoma. This is the arch hypodense lesion seen in the scene, almost completely replacing the wall better, which is heterogeneous enhancing with multiple non-contiguous enhancing lesion seen in the both lobe of liver. This is the case of GB mass with metastasis in the liver. Discussion, in my study, most common benign lesion of liver is hemangema. Most common primary malignant lesion is hepatocelular carcinoma. Most common malignant lesion are metastatic lesion. Metastatic lesion mostly comes from the GB mass, lesion primarily. Hepatocelular carcinoma commonly occurs in the hepatitis C patient. Mostly, hepatocelular carcinoma is associated with the chronic liver disease and raised alpha vitro protein. Hepatic hemangema typically demonstrates the peripheral nodular enhancement in the early phase followed by the centipital pattern of the fielding during the late phase. Hepatocelular carcinoma leads to peripheral enhancement that replace the large subcutaneous feeding vessels with centipital pattern of enhancement. Focal nodular hyperplasia. Hepatocelular carcinoma, administration of the liver is specific. Melodrium wastes the magnetic resonance contrast agent produced rapid enhancement of focal nodular hyperplasia due to arterial blood supply, jointing in a hypotense lesion on early films. On delays image, it becomes more iso-intensive with respect to normal liver. The central scar enhance on delays image as contrast gradually diffuse into the fibro-center of the mass. Hepatocelular carcinoma typically demonstrates a non-ream arterial phase hyperenforcement related to the liver panchema. Inter-hypertic colonio carcinoma, there is a peripheral enhancement throughout the both arterial and venous phase in liver metastasis. enhancement pattern of the liver metastasis varies depending on the primary magnetic. Metastatic liver lesion from the colon, stomach and pancreas usually show lower attenuation in contrast to brighter surrounding liver panchema on multifagicity of the liver. Hyperbascular metastasis such as those from the near undeclarent tumor, intensive carcinoma, breast carcinoma, valentina, and thyroid carcinoma appear as rapidly enhancing lesion which will on the arterial phase of enhancement. Conclusion. Multi-phase contours enhancing CT finding that help differentiate among the lesion include the lesions pattern of the vestibular enhancement and washout after contours and administration, the number of lesion and assisted finding in the liver. This is the references. Thank you.