 Hi everyone, my paper is on city finding in a case of mylidosis. This is a case report and Dr. Sarvesh, a junior resident from the Department of Rated Diagnosis from Arupati Medical College, Puducherry. I did it under the guidance of Dr. Nathish, HOD and professor of my department. So mylidosis is a mylidosis is caused by Burkwoodia pseudomonale. It affects multiple organs and mostly affected organ is the lung and it is followed by the spleen. It has a radiological features which is a mimico of TB and is being commonly misdiagnosed as TB in India. In patients with septicemia or fever of unknown etiology high suspicion is required for the diagnosis. We could present a pictorial review of the radiological manifestation in a patient presenting with multi visceral abscess which will help the radiables to arrive for an early diagnosis. So 46 year old male patient who came with the complaints of abdomen pain and five days of fever. The patient had an history of alcoholic liver disease and chronic calcific pancreatitis. The patient was known type 2 diabetes and was incident for 14 years with poor control of diabetes. The patient was evaluated for dengue, typhus, malaria and the results are negative. There were no history of TB or any history of contact with the TB patients for the patient. So the lab investigation with total count was 6700 and then your trophils were 86% and lymphocytes were 13. And ESR 90 and serum biliribin was 3.5. The HP was 10 and STP T, SCOT and ALP or everything was raised in this patient. So we took a chest X-ray in which the chest X-ray behavior which showed two ill-defined homogeneous rounded opacity in the left lower zone. There were no evidence of pleural effusion and there was no medial steenal widening and high blood opacity. Which are not seen in this patient. Then the patient had an undergone for a new SC and then new SC and doctor evaluation which showed multiple hyper-echoic lesion in both lobes of the liver which was less than one centimeter in diameter. There was spleen omega-3 with the multiple hyper-echoic lesions. Further the prostate was enlarged with a heterogeneous parankema and showed multiple hyper-echoic to an anarchy carriers within which was a suggesting and evolving axis. The lesion did not show any color uptake on a color Doppler and we were just suspecting for tuberculosis and melidosis. Then the patient was taken up for un-CECT in which the un-CECT the liver showed multiple solitary and multi-lopulated periferantly enhanced fluid density lesions in both lobes. In which the largest in the segment seven which was measuring for 25 into 21 mm. The few lesions were noted in the sub-capsular location with extension to perihepatic lesion. The lesion showed characteristic of enhancing internal separation which were presented as a cartwheel or a honeycomb pattern which you could see in this images. And this we had a multiple hypodensity in the spleen with melidosis and there was a spleenic abscess. We could see the spleen is mildly to moderately enlarged in size. And few hypodense lesions are seen in the gastro spleenic space. And in this we could see in multiple multi-lopulated enhancing hypodense lesion are found noted in both the kidneys with few of them were having pericapsular extension. Which we should make it clearly in this image. And then we took an accident coronal and sagittal segment of CECT of abdomen which shows multiple ill-defined periferally enhancing collection in the prostate. And the prostate was enlarged in size and showed people multi-lopulated periferally enhancing hypodense lesions. So in the blood culture which showed this Bercal triasura monally and so they treated with IV antibiotics with Cephasimide and Quaterrimaxazole for two weeks and then followed with an eradication therapy with Quaterrimaxazole for six months which was embarrassed. So the discussion is the myelodosis is endemic to the North and Australia in Asia. The nano-incidence of myelodosis is 12.7 in 100,000 in tropical countries. And the patient at risk are those who are exposed to wet soil and patient with diabetes, chronic liver disease and other immunocompromised conditions. Because of the similarity in clinical and radiological manifestation, it is usually mistaken for other infections like TB. Pheumonia is one of the most common clinical presentation. Lung is the most involved organ in radiological features of small nautiler densities which predominantly involves the upper lobes. Diagnosis, it is sparing the lung apex and presenting with the concurrent liver and sphenic abscess which helps in the diagnosis of myelodosis from tuberculosis. The mediasthenal lymphadenopathy and pleural diffusion are usually seen in tuberculosis and not in myelodosis and this helps in differentiating it from tuberculosis. Burghulia suromonale is the most common causative agent found in the patient with spleen, liver abscess and confirming the organism and blood culture is indefinite to diagnosis. So these are my references. Thank you.