 Hello everyone, I am Dr. Priyanka, finally a resident at Atal Bihari Vajpayee Institute of Medical Sciences and Dr. Ram Manohar Dhoiya Hospital, New Delhi. The topic of my paper is Role of multi-detector CT in evaluation of pancreatic lesions. The aim of my study is to detect and characterize pancreatic lesions by CT morphology. Multi-detector CT is the imaging modality of choice for pancreatic lesions. It allows multi-phasic imaging of pancreas, thus enabling excellent characterization of lesion. The pancreatic lesion can be classified into inflammatory condition, developmental anomalies, neoplasms and trauma. The pancreatic inflammatory conditions include acute chronic and autoimmune pancreatitis. Developmental anomalies include pancreatic divisive, annular pancreas, pancreatic head lobulation, a genesis of pancreas. The neoplasm includes pancreatic adenocarcinoma, endocrine tumors, intra-pancreatic meds, cystic lesions, etc. The infiltrative lesions, metabolic and others, include hemochromatosis, cystic fibrosis, fatty pancreas and heterotropic pancreas. This study was conducted in the Department of Radio Diagnosis at Atal Bihari Vajpayee Institute of Medical Sciences and Dr. Ram Manohar Dhoiya Hospital, New Delhi. It was conducted from 1st January 2021 to 30th June 2022 over 65 cases. These were the patients who presented with clinical features suggestive of pancreatic involvement or were diagnosed with pancreatic pathology on lab binding or USJ. The patients underwent pancreatic protocol on 128-slice CT scanner. The results were the maximum number of cases that were reported were of inflammatory etiology followed by neoplasm. The maximum number of neoplasm were of adenocarcinoma. Now the cases. The first case shows a bulky head of pancreas. The pancreatic body is replaced by a well-defined round homogeneous lute collection with thin enhancing wall-on-post-contrast images. There is no evidence of any internal necrotic composition suggestive of pancreatic pseudosist. Second case, there are axial and coronal reformated images on non-contrast pancreatic and portal venous phases in a case of gall-induced pannecrotizing pancreatitis. In this case, the pancreas is replaced by multiple peripherally enhancing thick wall collections in the head and tail region of pancreas. There is extensive surrounding fat trending findings are suggestive of world of necrosis. In this case, the pancreas shows diffuse atrophy with prominent main pancreatic duct, multiple punctate calcific foci which are seen along main pancreatic duct in the head body and tail of pancreas. These are suggestive of chronic pancreatitis. This is axial and coronal reformated images on non-contrast pancreatic and portal venous phase. In this case, an ill-defined heterogeneously enhancing lesion with predominantly high-pudence area is noted involving the head, insinid process and the proximal body of the pancreas with surrounding fat trending. The pancreas and the pancreatic and the common bile duct are dilated suggestive of mass forming pancreatitis. Case number five, the axial images in non-contrast arterial and venous phase in a case of blunt trauma. There is a linear high-pudence tract seen at the neck of pancreas with surrounding peripancreatic collection which is communicating at the site of leceration and is seen extending up to the spenic hyla. These findings and the view of history are suggestive of pancreatic leceration. Case number six, this is a post-contrast axial images which shows part of pancreas extending in the entero-lateral direction to second part of teardrum which is partially encircling that with narrowing of second part of teardrum and mild proximal diurnal dilatation. These findings are suggestive of annular pancreas, a developmental anomaly. Case number seven, this demonstrates pancreatic divism. This is a post-contrast coronal reformated images showing separate opening of dorsal and ventral pancreatic duct in the minor and major papilla respectively with no intercommunication between them. The dorsal pancreatic duct appear dilated due to involvement of the minor papilla ventral pancreatic duct and the common bile duct are opening into major diurnal papilla which appear normal in caliber. Case number seven, it demonstrates dorsal pancreatic agenesis. These are post-contrast coronal reformated images of two different cases. In both the cases there is non-visualization of the pancreatic neck, body and tail. However, head is visualized and it appears normal in morphology, attenuation and post-contrast enhancement. Now the neoplasm. This slide shows non-contrast pancreatic and venous phase axial images showing heterogeneously hypo attenuating solid mass region with cystic or necrotic areas in the region of head and proximal body of the pancreas causing obstruction of main pancreatic duct leading to its dilatation and pancreatic atrophy with central and peripheral obstructive obelopathy. These findings are suggestive of pancreatic adenocarcinoma. Again, this is a case of pancreatic adenocarcinoma. In this there is an ill-defined, mildly enhancing, high-pudence lesion. In the tail of pancreas with distal MPD dilatation and atrophy of pancreatic tail, there is no evidence of gasophic foci within the lesion or in the MPD. Lesion has clear fat plane with stomach, transverse colon anteriorly and bilateral kidneys posteriorly. This case demonstrates musinocyst adenoma. Non-contrast pancreatic and venous phase axial images are shown which shows well-defined, lobulated, thick walled peripherally enhancing homogeneously high-pudence lesion involving insinate process. There is no obvious communication with the MPD scene. These findings are in favor of musinocyst adenoma. Case number 11, non-contrast pancreatic portal venous and delayed phase images of axial as well as coronal reformated images shows multi-loculated, multi-cystic lesion of flu density in the head of pancreas and insinate process with few dilated side branches. The lesion is orbiting IVC. The lesion is also orbiting diodenum enterolaterally. Findings are suggestive of IPMN. Case number 12, non-contrast pancreatic and venous phase coronal images shows multi-loculated, cystic lesion of fluid intensity in the region of head of pancreas and insinate process suggestive of cirrhosis adenoma. Case 13, which demonstrates a spin that is solid pseudo-pepillary epikellium neoplasm. In this, we have a well-defined, lobulated, heterogeneously high-pudence solid mass lesion arising exorphatically from the head of pancreas and showing heterogeneous enhancement with multiple non-enhancing areas which are suggestive of necrosis or cystic component. There are multiple profile of calcification seen within the lesion. Now, the last case, which is of neuroendocrine tumor of the pancreas. This slide shows non-contrast pancreatic, total venous and dilate phase axial as well as coronal images. In these images, there is evidence of a well-defined homogenous, isodense mass lesion in the head of pancreas and insinate process with few calcific foci within the lesion. There is hyper enhancement in the early arterial phase followed by reduced enhancement and washout in the subsequent late arterial and venous phase images. The lesion was supplied by gastroduronal artery. It is causing dilatation of empathy in the body and tail of pancreas with abrupt cut-off at the periphery of the mass. CBD appears prominent with abrupt cut-off at the periphery of the mass in the head of pancreas. These findings are in favour of neuroendocrine tumor of the pancreas. So, in our study, various pathologies were detected. Pancreatitis showed male predilection in age group 31 to 40 years with alcohol abuse being the common entheological factor. Pancreatic neoplasm showed male predilection in the elderly group and commonly affected the body. Pancreatic duct and CBD dilatation was noted in the case of pancreatic neoplasm. Of the total cases of pancreatic neoplasm, solid mass lesions were more common with adenocarcinoma being the common entity. Few cases of developmental anomalies in blood trauma were also being reported. Hence, multi-detector CT plays an important role in the detection and characterization of pancreatic lesion. It can determine the extent of the lesion. It can demonstrate the site of metastasis and the vascular involvement. Thus, playing a pivotal role in determining the management and prognosis. These are the references. Thank you.