 So go by the duration, acute or chronic, more than chronic, more than 40 tasks don't, don't wait, in long term you can only manage this diet. So acute pulmonary edema, again, or dialysis patients or chronic indie patients come commonly with dialysis, it to the ER, well a very, you know, somebody with urea of CED patients, urea of 80 or 100, okay, creatinine of 3.5, coming with pulmonary edema, I see a lot of people giving lasix 20 milligram or 40 milligram. So don't do that. When you give a lasix, the thumb rule again I'll tell you, if somebody is having the urea of an 100 or urea of an 80, at least make sure you give 80 milligram of lasix. This is a thumb rule what we tell, so if somebody is having 100 urea, 120, at least think about even 100 milligram. So 20 or 40 will not help you in any way, it's not even going to touch you. So approximately what about the solutes urea you'll have, you know, you need one lasix to bind to one urea you take like that. So one lasix will bind to one urea, at least like that thumb rule, you know, you think like that and then you give lasix. So whenever we'll come with severe pulmonary edema, at least give, if somebody's creatinine is 3.5 or 40 urea of 100, just give 80 to 100 milligram. Once he stabilizes, then you put him on infusion at 10 milligram per hour or something like that. If they don't respond to lasix, then we only we dilate them. And also in a year, don't hesitate to intubate them. So you will in a respiratory distress, don't wait, you know, the moment you wait longer time is also, what we do is just sedate or paralyze and intubate them. Once you do that, you know, they do really well because we'll lose a lot of patients waiting for them, you know, for the lasix to act or in IV and then, you know, we normally severely breathless. We don't wait, we sedate them, put them on ventilator, then we continue. It's much safer to do it. So I'll just give them some brief thing, whatever I could do with all the nephrological emergency. So if you have any questions, I'll take it all depends see, see normally what happens is, you know, I've told you how much of the loss in case the potassium is around 3 is at least 1115 to 200 milligrams of potassium loss. I told you, you know, you give 20, 15, only around 20 ml, three times a day you give, you are only giving it for, you're giving 60 milligrams per day. So at least, you know, 60, 60, 60, okay, at least two to three days, you have to give that bottle of whatever you give for three things. So at least 20 ml, three times a day, you need for two days or three days. But again, you have to repeat after two days. It's not exactly, you know, you do, you can't exactly, that is a loss. It is overall, it can underestimate or overestimate. But you know, don't, if you're giving all those things, make sure you repeat the test again after two days. That's very important. Sir, in selected few groups, you said the diuretics and hyponatremia, will you please elaborate that group? Sir, say that's right. Hyponatremia always, people think only the salt is there, the sodium is low. No. Always, it's problem with the water. Hyponatremia is a problem related to water. It is not related to the salt. So whatever, what happens to the water, when somebody is clinical, you will be because always the water is extra. So when you clear the water, automatically the sodium improves. So that's our role of diurutin. That's our role, we will give diurutin because it helps somehow with 3 percent saline, you give diuretic also, it helps you to automatically remove the extra water from the body. That's why in SAIDH or all these eulemic hyponatremia, we restrict the water also. So what happens is, you just restrict less than 1 litre, automatically chronic hyponatremia who has SAIDH, the only treatment is salt and water restriction. Means salt, normally they give more salt, 8 to 10 grams of salt. But water we restrict around 1 litre. So again, eulemic, again the salt. So the hyper eulemic, salt-free fluid restriction, hyper eulemic, salt-free fluid restriction. Eulemic, more salt, less water and depletionally you just, that doesn't matter. I can talk but SAIDH is there, it's going to talk and SAIDH talks, I leave it to me, he's the best person. So I leave the urology, obstructions, stone, everything to SAIDH, urology. It's not like I'm washing my hands but because my expert colleague is there to talk on that emergency, I leave that to him. No, I'm sorry, okay. So I didn't, I didn't listen to your questions, sir. If you don't mind. Regarding kidney urinary stones, any obstruction can cause acute kidney injury. Up to what size of the calculate we can... So SAIDH doesn't matter. See, the urilateral obstruction will not cause AK high. We have two kidneys, one kidney obstruction will not lead to AK, unless that is causing infection or something. For somebody to have AK high, we should have bilateral obstruction which is very, very rare. Or a single kidney with obstruction. Somebody has a single kidney and stone causing obstruction can cause AK high. But just because somebody has a, whatever stone, less than 5 mm will not cause idonophilus. 5 to 10 mm or more than that, in the PUJ junction, idonophilus, it will not lead to AK high. But you know, the problem is because of obstruction, whether he has urinary infection because of that or pyonophilus, whatever is the, that's equally cause AK high. But urilateral obstruction will not cause AK high at all, whatever is the size of the stone. Another comment, it was a very comprehensive coverage of multiple conditions. I just want to say for the benefit of any junior doctors and practitioners, correction of hyponatremia, they need to be cautious because rapid correction can lead to severe demyelination. Correct. That's the reason I told, we probably don't correct more than 8 to 10 minutes per day. But in case it's very, very acute or severe patients in seizure, then up to 10 to 12 also. But don't correct more than that. It can cause central demyelination, chronic demyelination. My question is different. The patients who are on hemodialysis, the skin color changes. What is the exact reason? So this is out of my thing but still, it's a uremia itself. You know, uremic toxins, it can cause uremic frost, uremia, the toxins itself, lot of things, skin color can change. Is it correct? The urochrome will not be removed. If you ask me, there are so many things. There are so many things. Small molecules, middle molecules, larger molecules, so many molecules which are there. It can remove certain molecules. It can't remove all the molecules. So these things are the one. There are so many other toxins which are there, which causes uremia and all these color changes. And one more question for Dr. Harsita, which I wanted to ask. Probably you can answer that question. Allergy. Regarding allergy, we have treated in a hospital setup for an allergy with all the drugs. So in between what happened, the patient survived, everything is okay. But he was hitting his heart with a fist during that treatment. What may be the reason? I don't know, sir. I don't know why he was hitting his heart. I don't know, sir. I don't know why he was hitting his heart. I don't know. I don't know. I don't know. Why he was hitting his heart? I don't know. I don't know. I don't know. Probably... No, he has treated allergy recovered. With a fist. Both the heart. I don't know. I just want to know the answer. Probably even I don't even ask Dr. Harsita about him. I said, as long as he is not hitting us, I am happy. Probably even I don't even ask Dr. Harsita about him. I said, as long as he is not hitting us, I am happy. Anything else or shall I hand over to my next speaker? Because... Okay, thanks a lot. Dr. Shikant Rao, to hand over a moment to Dr. Satish M.