 Doctor, everyone. Today, myself, Dr. Oslo Shahan, I'm here to present a case report on a very rare case of 200 gut losses. So a 13-year-old girl presented to our institute with swelling in the bilateral hip region over a period of two years duration. The swelling increased progressive and it was associated with pain. A medical history was insignificant. There were no in-history of trauma, nemochuria, abdominal pain, renal insufficiency, or similar family history. Physical examination revealed tender, hard mass around both the hip joint. The masses were not attached to the underlying bone. The temperature and texture of the overlying skin was not normal. We then subjected the patient to some biochemical investigation, which revealed that the serum calcium, serum alginin, vitamin D, liver function test, renal function test were not normal. However, the phosphate level were high in the patient. It was approximately around 7.2 milligram per deciliter. She had mild lymphocytosis, with phrase erythrocyte sedimentation rate, which signifies that the patient was suffering from a chronic inflammatory process. The patient was sent to the radiology department for further investigation to characterize the patient. When she came to our department, the first investigation that we did was an accident. We looked at the accident, which revealed that there were large bilateral amorphous soft tissue, multi-locular calcification, seen around the hip with internal cystic and sedimentation level. The underlying bone structures were, in fact, were, in fact, unremarkable. We then performed USC of both the hip region, which we were hypoechoic heterogeneous solvage measure approximately 8 into 4 centimeter in the upper lateral part of the thigh region on the left side and similar on the right side, with multiple large forbears of calcification, the peripherally as well as centrally in the subcutaneous as well as musculoskeletal. The patient was then sent to the patient for acid investigation, which revealed bilaterally follows symmetrical in shape and size, predominantly calcified salt. Noted in the subcutaneous and muscle, the plane of bilateral gluteal region with multiple cystic areas containing dense fluid-fluid level and enhancing areas in this lesion. Few feeding vessels were also noted in this lesion. We then subjected the patient to MRI finding. These were the images that were on tent. These were the T2-FAC-7 images. On MRI, there were periarticular multi-locular calcified muscles with significant fluid-calcium level involved in bilateral gluteus maximus muscle with soft tissue extension around the hip and the upper pibus. It showed heterogeneous T1 signal intensity and predominantly high T2 signal intensity with multiple calcium fluid. Within performed a u-cigarette-repenis examination of the lesion, which revealed the microscopic examination of the lesion involved, revealed smears where, in fact, our cellulome only composed of calcified material deposit. There was no necrosis, no granuloma. Minus. The mass was physically removed, and the patient was kept on phosphate-restricted diet then after. The tumoral calcinosis is, in fact, a rare and benign pathological condition, which is characterized by a calcified deposit of hydroxy, hepatite, or amorphous calcium, phosphate crystals, in the periarticular-connected tissue. They are not true neoplasms, and they do not pose as vital excels. Despite several theories, exact pathogenesis of the condition remain, in fact, unclear. In terms of its epidemiology, the idiopathic form usually occurs in the first three decades of life, no appearances, sex predominance, and an apparent criticalination towards black population roster. On clinical evaluation, it presents as a slow-growing soft tissue mass, painless in most cases, arising near large joints with the hip joint being the most common site in the reported series. The other site are elbow, shoulder, knee, wrist, hand, and foot. The differential diagnosis, considered in this case a soft tissue calcification and fluid calcinosis, secondary to renal failure, which can be differentiated by the renal pulsatist and vitamin D-lib. Sinable contromatosis, which is usually intraarticular as soon as ring arc appears. Myocytus ossificens, which is characterized by rapid evolution and relaxed lobular morphology. Complication related to this condition includes pain-neutral nerve compression, ulceration, bacterial infection, and cosmetic dysregulation. Simple complication, severe complication is very rare, and it will influence loss of vision. The treatment choice is surgical protection as the use of medical treatment for hypophosphatemia is controversial. Steroid and radiation use were also suggested by other authors. However, complete surgical removal to prevent reoccurrence is, in fact, the best approach. Conclusion, tumoral calcinosis, though a rare condition, should be considered a possible diagnosis in the ASDU and radiographic picture, as it is not a typical neoplasma, or a phony tumor. The decision to intervene is entirely based on the patient's disability and complication as a result of the pathogenosis. The treatment remains essentially surgical, and the prognosis is usually lost. These were the differences that I obtained from. Thank you.