 Abstract bisphenol F, BPF, has been introduced as a replacement for bisphenol A, BPA, in the production of consumer goods due to its endocrine disruption properties. However, BPF monomers can also be released into the environment and enter the fruit chain, leading to human exposure at low dosages. This exposure can have detrimental effects on the liver, which is particularly susceptible to such low dosages. In this study, researchers evaluated the effects of BPF administration on oxidative stress in the livers of lactating rats and their offspring. They found that BPF caused increased oxidative stress in the livers of lactating rats and their offspring, indicating that BPF should not be used in consumer products without further investigation. This article was authored by Beatriz Linillos-Prodillo, Lisa Rancan, Julio Garcia-Muriz, and others.