 The presence of the toxin beta-n-methylaminoel alanine, BMAA, in certain environments has been linked to the development of neurodegenerative diseases such as a myotrophic lateral sclerosis, ALS, Alzheimer's disease, and Parkinson's disease. This toxin can be found in cyanobacteria, which are common in freshwater bodies around the world. Cyanobacterial blooms have been observed in many areas where ALS cases are high, suggesting a possible connection between the two. Additionally, BMAA has been shown to induce oxidative stress and damage to the mitochondria, both of which are known contributors to neurodegeneration. Furthermore, BMAA has been shown to directly affect the NMDA and AMPA receptor systems, which are important for normal neuronal function. These findings suggest that BMAA may be responsible for the development of ALS and related neurodegenerative diseases. This article was authored by Elijah W. Stommel, Tracy A. Coller, and Sandra A. Bannock. We are article.tv, links in the description below.