re Re: ERV's in chimps and gorillas but not humans

In population genetics, fixation occurs when every individual within a population has the same allele at a particular locus. The allele, such as a single point mutation or whole gene, will be initially rare (e.g. originating in one individual), but can spread through the population by random genetic drift and/or positive selection. Once the frequency of the allele is at 100%, being possessed by each member, it is said to be "fixed" in the population

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[demer64]

At o:13 its says chimps are the most closely related, not related.

[Ishta5]

we are all related but they are our closest relatives, the point is that this was a response to a video. It used this paper as a source for how we are not related and that there is no common ancestor

[Desertphile]

Thank you: I just had this explained to me in talk.origins and I'm rather ashamed to see how stupid I was not to know this. It explains why Humans, for example, do not have some ERVs that Chimps and Gorillas share (at the same insertion point) even though our common ancestor with chimps and gorillas did.

[MadHatterTruth]

poor rtard

[phoenixshade3]

You may choose to remain ignorant if you wish, but it's not due to lack of evidence; only to your inability to recognize a millenia-old book of mythology for what it is, combined with a truly stunning lack of intellectual curiosity and critical thinking ability.

[demer64]

The only relaive thing about humans and chimps is they were both created from the soils of the Earth.

[Bucketses]

See that there are two ERV's that are not present in the entire population, so they are obviously inserted, and like we know, starting though only one person. There is no evidence that it is beneficial, but there is evidence that it is potentially a causer of disease, so how can these ERV's be so widespread yet not helped through natural selection? Neutral drift. Neutral drift can fix alleles differently in different populations.

[Bucketses]

Also see the abstract of this paper:

Insertional polymorphisms of full-length endogenous retrovirusesin humans

Geoffrey Turner, Madalina Barbulescu, Mei Su, Michael I. Jensen-Seaman, Kenneth K. Kidd and Jack Lenz

[Bucketses]

Not all genes have the same history so fixation is not expected to happen all at the same time. If you check the Punnet square for 0i you will find that 50% are 0i's and the other 50% 00 and ii together so there is no statistical advantage to 0i, fixation will depend on random factors that affect the population. This means I would have to retract a previous statement done here I believe.

[Bucketses]

Neutral genetic drift promotes homozygousity and can preserve non beneficial alleles and cause them to dominate. Fixed ERV's on gorillas and chimps and not in humans is not controversial. There is absolutely no problem here. Here is an interesting paper on genetic drift:

Genetic drift at expanding frontiers promotes gene segregation.

Oskar Hallatschek, Pascal Hersen, Sharad Ramanathan, and David R. Nelson

Size fluctuations and events in the population can change what alleles get fixed.

[Bucketses]

If the pattern did not exist you could not build the pattern. What you are is like saying that periodicity of the table of elements is just an interpretation, there is no periodicity, only similar properties. Well, we can actually measure the properties and show that they repeat the same way that we can pinpoint what are the features that define the groups within groups. Patterns in the data are properties of the data. All scientific theories are constructed from patterns in data.