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I THINK ALL THESE STUDIES ARE FAULTY AND ARE USING INCORRECT REASONING. They are wasting their money to fund this kind of research. I might be wrong, but follow my line of though.
AngII is a very potent vasoconstrictor. This vasoconstriction, the RBCs tear through the tunica intima causing the anuerysm. The clotting mechanism are activated , therefore artheriosclerosis.
It is due to constant vasoconstriction and not the Angiotensinogen II ,that you get a larger AAA and artheriosclerosis.
Christom88 2 years ago
I THINK ALL THESE STUDIES ARE FAULTY AND ARE USING INCORRECT REASONING. They are wasting their money to fund this kind of research. I might be wrong, but follow my line of though.
AngII is a very potent vasoconstrictor and this vasoconstriction causes the RBCs to be in constant contact with the tunica intima causing tears. The tears cause the release of clotting mech.
It is due to constant vasoconstriction and not the Angiotensinogen II ,that you get a larger AAA and artheriosclerosis.
Christom88 2 years ago