Muhammad Aziz, a Kurdish victim of 1988 Gas attack of the city of Halabja, Iraq,He has 100% disability due to his severe chronic lung disease,He is in an urgent need of medical treatment because of injuries to his respiratory system. due to the inhalation of chemical gases during the bombardment of Halabja on march 16 1988 He is a candidate for lung transplant but the waiting list is long and it may become too late for him when one is ready because of the fast deterioration of his lungs
/watch?v=l3K4oweyDB0 Following this link (copy and paste it in Youtube search) you will find one of the largest COPD community. I found there a lot of tips, answers about COPD. I hope it will help you. Join and help it's free :)
Consider switching to a Smart Nasal Cannula w/ Oxyview from INGEN Technologies. It's inline flow-meter offers patients peace-of-mind and worry-free wear by accurately displaying the Lpm rate of oxygen.
centre as it is significantly higher! so does 02 remove the stimulus to breath or is it CO2 narcosis (due to hypercpania) that depresses the central nervous system and ultimately the respiratory centre? anybody know? thanks
At a P02 of 60mmhg peripheral chemoreceptors are stimulated, so it has been said that COPD patiens rely on there hypoxic drive more than there hypercpanic drive to breathe. However during an exacerbation ventilation is impaired and CO2 can rise, and would giving high conc oxygen only increase hypercapnia by mechanism of the haldane effect and Hypoxic pulmoanry vasoconstriction. the patient is adequately oxygenated but should the high CO2 not reach a threshold which stimulates the respiratory
can anyone shed some light on this for me..... Is there any hard evidence for the removal of the hypoxic drive by administering high flow oxygen? My current understanding is that in type 2 resp failure patients become hypercapnic so understandleable that there normal CO2 range will be chronically increased, there there may be decreased central sensitivity in the Medulla as acidity is buffered by bicarb reasbsorption ect ....(see next comment)
I've been pimped on this question by internists, etc and they say what you say...O2 mediated decrease drive as the explanation for the lunger as to why they get hypercarbic?? WRONG! at least mostly wrong...
What happens is at baseline these pts have destructed alevelor units that are in a sense not functional. What the lung does is vasoconstrict to shunt blood to good lung and 100% o2 dilates these vessels to create an intrapulmonary shunt to bad lung and co2 goes up. (PGY-1 Gen Surg)
if co2 high, csf will be acidic,bicarb normals csf ph,that is why cental receptors dont work. peripheral receptors stimulate to breathe at sat of 90%. unless c02 is 55 or higher they are not a retainer and there is little concern because central receptors will work. if sat is high peripheral receptors dont work. it depends on hydrogen ion concentration of csf how well central receptors work.( RRT educator.)
The explanation of the histology of emphysema is not correct. The pictures shown at minute 1:13 are NOT the vasculature. They show the loss of the alveolar walls, which is the hallmark of emphysema.
wow, this is the 4th video of yours that I have watched... you are a natural teacher. GREAT GREAT GREAT job
ktagliam 4 days ago
Comment removed
drericstrong 6 months ago
This has been flagged as spam show
Muhammad Aziz, a Kurdish victim of 1988 Gas attack of the city of Halabja, Iraq,He has 100% disability due to his severe chronic lung disease,He is in an urgent need of medical treatment because of injuries to his respiratory system. due to the inhalation of chemical gases during the bombardment of Halabja on march 16 1988 He is a candidate for lung transplant but the waiting list is long and it may become too late for him when one is ready because of the fast deterioration of his lungs
halabjakam88 6 months ago
Thank You! :)
facingeast1 9 months ago
/watch?v=l3K4oweyDB0 Following this link (copy and paste it in Youtube search) you will find one of the largest COPD community. I found there a lot of tips, answers about COPD. I hope it will help you. Join and help it's free :)
eddieas23 9 months ago
Superb, this makes it all really clear. Thanks.
wudgee 10 months ago
this was the most helpful explanation I have ever heard. Thank you so much.
cyun822 11 months ago 2
Awesome video. Great explanation, great energy, great visuals. Can't wait to explore more vids of yours now.
jilleeeeee 11 months ago
You Rule Dave
dwe253 1 year ago
Consider switching to a Smart Nasal Cannula w/ Oxyview from INGEN Technologies. It's inline flow-meter offers patients peace-of-mind and worry-free wear by accurately displaying the Lpm rate of oxygen.
ckaminsk 1 year ago
centre as it is significantly higher! so does 02 remove the stimulus to breath or is it CO2 narcosis (due to hypercpania) that depresses the central nervous system and ultimately the respiratory centre? anybody know? thanks
stethorbs10 1 year ago
At a P02 of 60mmhg peripheral chemoreceptors are stimulated, so it has been said that COPD patiens rely on there hypoxic drive more than there hypercpanic drive to breathe. However during an exacerbation ventilation is impaired and CO2 can rise, and would giving high conc oxygen only increase hypercapnia by mechanism of the haldane effect and Hypoxic pulmoanry vasoconstriction. the patient is adequately oxygenated but should the high CO2 not reach a threshold which stimulates the respiratory
stethorbs10 1 year ago
can anyone shed some light on this for me..... Is there any hard evidence for the removal of the hypoxic drive by administering high flow oxygen? My current understanding is that in type 2 resp failure patients become hypercapnic so understandleable that there normal CO2 range will be chronically increased, there there may be decreased central sensitivity in the Medulla as acidity is buffered by bicarb reasbsorption ect ....(see next comment)
stethorbs10 1 year ago
RT's, nurses, internists, medical students all dont get it...but...a pulmonologist will explain if you ask him if he has a half a brain
mstrode4449 1 year ago
I've been pimped on this question by internists, etc and they say what you say...O2 mediated decrease drive as the explanation for the lunger as to why they get hypercarbic?? WRONG! at least mostly wrong...
What happens is at baseline these pts have destructed alevelor units that are in a sense not functional. What the lung does is vasoconstrict to shunt blood to good lung and 100% o2 dilates these vessels to create an intrapulmonary shunt to bad lung and co2 goes up. (PGY-1 Gen Surg)
mstrode4449 1 year ago
@mstrode4449 I thought high o2 in the lungs causes vasoconstriction of the vessels.
koimaster 1 year ago
thanks for your videos! helps me a lot in preparing for our local board exam in the Philippines..
RNfermanes 1 year ago
if co2 high, csf will be acidic,bicarb normals csf ph,that is why cental receptors dont work. peripheral receptors stimulate to breathe at sat of 90%. unless c02 is 55 or higher they are not a retainer and there is little concern because central receptors will work. if sat is high peripheral receptors dont work. it depends on hydrogen ion concentration of csf how well central receptors work.( RRT educator.)
gblespaul 1 year ago
thanks so much! ^^
kingCRAM11 1 year ago
Doesn't a high rate of Co2 promotes a change in PH?
gamekillanyc 2 years ago
@gamekillanyc Yes! It should make it acidotic. They are in an inverse relationship with each other.
AngelOne11 2 years ago
hey guys. how can i get the video here.
agalaites032109 2 years ago
Thank you so much!
AK2223 2 years ago
excellent Ed. Thank you
elijahdomingo 2 years ago
if you mean his name his Ed, i think you're wrong, =P
krisvincent 2 years ago
The explanation of the histology of emphysema is not correct. The pictures shown at minute 1:13 are NOT the vasculature. They show the loss of the alveolar walls, which is the hallmark of emphysema.
The remainder of the video is very good!
rpalmeri 2 years ago
Thank you! you are a great teacher! I wish you were my teacher during my nursing in class days.
Mydreydan 2 years ago
Great Video!
isfahan1988 3 years ago
Thank-you for explaining a complex process simply.
harbor913 3 years ago