Would the treatment for this baby's case similar to that of Tetralogy of Fallot? If so what do you think the treatment should be and when it should be done? Thank you.
Examinations were therefore performed and it was found out that she has got heart problems which is very similar to what you are describing here, WITHOUT the overriding aorta (because from the echocardiogram result, it seems that the aorta is correctly positioned over the left ventricle).
In summary, she has got MEDIUM pulmonary stenosis, MEDIUM vsd, MEDIUM right ventricular hypertrophy, and TINY atrial septal defect.
Hi JPShand, thanks for uploading this excellent video. I have a couple of questions that I hope you would be able to help me with.
A baby (only a few days old) is found to turn blue in her lips and face, ONLY when she cries. During other conditions, her color is normal reddish (with NO PROBLEM in feeding and NO rapid/short breathing).
Hello you were right the first time about etiology. According to first aid 2011 and Robbins and Cotrain Pathlogy its anteroSUPERIOR displacement of infundibular septum.
What is the reason for the syncope episodes. If most of the blood goes up the aorta feeding the carotids, and the PA feed the lungs, would that cause dyspnea and only with severe decrease in O2 sats cause the episode.
The deoxygenated blood that is returning to the heart from the body (via the superior/ inferior vena cava) is supposed to be re-oxygenated by the lungs (via the pulmonary artery). But, it is not because of the stenosis. Instead, this deoxygenated blood is re-routed back to the body (through the aorta)! Alas, this deoxygenated blood cannot feed the brain the oxygen it needs, the reticular activating can't function without proper oxygenation and thus it shuts down, resulting in syncope.
About the hypercyanotic attacks that occur when crying, it happens because the sympathetic response causes an increase in the pulmonary stenosis (the pulmonary stenosis is actually a right outflow tract obstruction). Sympathetic stimulation of the heart actually worsens the right outflow tract obstruction, worsening the pulmonary stenosis and causing increased PVR, increasing the right to left shunt and causing them to become blue.
i have a question. i m a final year student. just wondering y would we give fuilds to the child? the right ventricle is already hypertrophied, shouldnt we be giving diuretics? n if not, y not? plz guide
What's the pharmacology mechanism of morphine in the treatment of the cianosis crises? Which dose should I use in mg/kg? Thanks. edufrancis@gmail.com.
lol, nice, i have a pathology exam tomorrow and i was looking for a right to left shunt video and i look at the uploading date... hahaha how lucky am i, if i had my exam a week ago i would have not seen this video, thx alot, a real great effort, thx
Nice! How does squatting prevent the overriding Aorta from getting the deoxygenated blood? Does it increase the pressure in the left ventricle and force blood into the Pulmonary artery? Is that what you meant by afterload?
Exactly! Increased afterload via crimping of the femoral arteries will increase the pressure against the LV ejection, and therefore push blood toward the area of less resistance (which upon squatting became the pulmonary artery). Before the squat, the path of least resistance was through the over-riding aorta.
Would the treatment for this baby's case similar to that of Tetralogy of Fallot? If so what do you think the treatment should be and when it should be done? Thank you.
OrangIndonesiaAsli9 2 weeks ago
Examinations were therefore performed and it was found out that she has got heart problems which is very similar to what you are describing here, WITHOUT the overriding aorta (because from the echocardiogram result, it seems that the aorta is correctly positioned over the left ventricle).
In summary, she has got MEDIUM pulmonary stenosis, MEDIUM vsd, MEDIUM right ventricular hypertrophy, and TINY atrial septal defect.
OrangIndonesiaAsli9 2 weeks ago
Hi JPShand, thanks for uploading this excellent video. I have a couple of questions that I hope you would be able to help me with.
A baby (only a few days old) is found to turn blue in her lips and face, ONLY when she cries. During other conditions, her color is normal reddish (with NO PROBLEM in feeding and NO rapid/short breathing).
OrangIndonesiaAsli9 2 weeks ago
Excellent video! Thank you!
Ositobobito 2 weeks ago
really comprehensive and explanatory idd.finally got some important points via this video what my professor couldnt teach properly enough
taurus35 3 weeks ago
thank you for your VERY thorough explanation! Do more videos of the heart :)
annette020 1 month ago
hola soy de Ecuador me encanto el video :)... Hi, Im from Ecuador really loved this video thak u so much, youre doing medicine easier...
karlalorena1302 1 month ago
Hi. Im just curious about the pathophysiology of CHD child who has severe malnutrition?
This video is great btw. :)
sordidfate 2 months ago
great vids!! may u explain the shifting position of cardiac apex in case of rt n lt ventricular hypertrophy ?igot confused a lil ::)
leojacj1 2 months ago
Thanks a lot
clarvery 2 months ago
thank you
mrpowell0981 3 months ago
Thanks a lot ,appreciate the help :)
amerribhi 3 months ago
Thanks!! Can you make a video on Ebstein Anomaly please?
prtysha07 3 months ago
This was really helpful. I completely understand Tetralogy of Fallot now. Please post more educational videos. Thank you, thank you so much!!!
KGilles100 3 months ago
you are amazing !!
wlaa2009 4 months ago
dude you're awesome! this helped alot, thanks so much! :D
nucleola 4 months ago
Hello you were right the first time about etiology. According to first aid 2011 and Robbins and Cotrain Pathlogy its anteroSUPERIOR displacement of infundibular septum.
HugAmango 5 months ago
Awesome explanation on Tetralogy of fallot!
YUSHIU5 5 months ago
very good & clear explanation! thanks!
Abster1911 7 months ago
Excellent explanation!! Better than my professors...kind of depressing since youtube is free.
spoekr8 7 months ago
This video explained all 4. Very good. Thanks
Malagu40 7 months ago
I am an adult TOF. This is great. I remember "squatting" as a child. 20 years ago I had total correction done.
jenniferw87 7 months ago
you're a friggin genius! you should seriously make more videos man this has made my life so much easier! Encore S'il vous plait <3
shahdehbomb 8 months ago
Spelled: hypertrophy
Pronounced: /hai-PER-truh-fi/
alanheadbloom 9 months ago
This really helped me learn ToF for my exams. Thank you!
RobertDavidStevenson 9 months ago
Thanks alot Dude, appreciate it!!
nodoctoryet 9 months ago
Thanks!
UberJerkyNerd 9 months ago
This was very helpful! Thanks for the mnemonic
kareecemoonasingh 9 months ago
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very nice video. Are you a doctor? I think you definately know what you are talking about.
were455 9 months ago
very nice video? Are you a doctor? I think you definately know what you are talking about.
were455 9 months ago
Certainly did learn something. One of the best explanations ever. Thank you!!
TheAnaRayAna 9 months ago
What is the reason for the syncope episodes. If most of the blood goes up the aorta feeding the carotids, and the PA feed the lungs, would that cause dyspnea and only with severe decrease in O2 sats cause the episode.
pdmc5521 10 months ago
Hello!
The deoxygenated blood that is returning to the heart from the body (via the superior/ inferior vena cava) is supposed to be re-oxygenated by the lungs (via the pulmonary artery). But, it is not because of the stenosis. Instead, this deoxygenated blood is re-routed back to the body (through the aorta)! Alas, this deoxygenated blood cannot feed the brain the oxygen it needs, the reticular activating can't function without proper oxygenation and thus it shuts down, resulting in syncope.
JPShand 10 months ago
Thank you for the question!
Everyone should feel free to ask any more questions you want answered here.
If anyone wants me to explain any other disease processes in a new video, please ask! I'll do it!
Or at least, do my best!
Thank you all for all your great feedback!!
JPShand 10 months ago
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pdmc5521 10 months ago
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pdmc5521 10 months ago
About the hypercyanotic attacks that occur when crying, it happens because the sympathetic response causes an increase in the pulmonary stenosis (the pulmonary stenosis is actually a right outflow tract obstruction). Sympathetic stimulation of the heart actually worsens the right outflow tract obstruction, worsening the pulmonary stenosis and causing increased PVR, increasing the right to left shunt and causing them to become blue.
anickad5 10 months ago
@anickad5 Absolutely, excellent addition! @pdmc5521 this helps answer the question as to why the syncope occurs during the tet spell.
JPShand 10 months ago
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rtjonesmd 6 months ago
Comment removed
rtjonesmd 6 months ago
i have a question. i m a final year student. just wondering y would we give fuilds to the child? the right ventricle is already hypertrophied, shouldnt we be giving diuretics? n if not, y not? plz guide
kblossomsk 11 months ago
Awesome explanation. Thank you.
llkwaigone 11 months ago
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What's the pharmacology mechanism of morphine in the treatment of the cianosis crises? Which dose should I use in mg/kg? Thanks. edufrancis@gmail.com.
edu0123456 11 months ago
Very well explained; good organization and coordination of information.
You made it easier to understand. Thank you sir! :)
blacklibra0175 11 months ago
Very well explained; good organization and coordination of information.
You made it easier to understand. Thank you sir! :)
blacklibra0175 11 months ago
very nice vid.thnks!
beardfetish 11 months ago
great explanation
XpiR3d 1 year ago
Finally I understood exactly for what I've been operated when I was 1 year old. Thanks!
Astonmartin4444 1 year ago
thank you very much..we just finished our case presentation awhile ago. this helps me a lot.. thank you so much!!
angel01lady 1 year ago
wow very nice thank u ...it helped me alot.... plz we want more amazing videos like this
run2271984 1 year ago
great video, I am a MD and it was perfectly explained
tomasgrullon 1 year ago
That was FANTASTIC!
OriginalMissKitty 1 year ago
I'm a nursing student and have a test on this in 2 weeks.This videos is wonderful. please continue to make more!!!
summereve18 1 year ago
please explain other things, you are talented in teaching :)
sweet1991revenge 1 year ago
I really like this explanation. I am a student as well, and have a test on Congenital Heart Defects this week. Helped a lot! thanks!
Fontaineiii 1 year ago
Great explanation, helped me immensely in trying to understand tet spells. Thanks for the great vid!!!
bigsella86 1 year ago
THANK YOU SO MUCH!!!
teachmeanatomy 1 year ago
a little boring but good effort..
spicyjampong 1 year ago
lol, nice, i have a pathology exam tomorrow and i was looking for a right to left shunt video and i look at the uploading date... hahaha how lucky am i, if i had my exam a week ago i would have not seen this video, thx alot, a real great effort, thx
BabylonLynx 1 year ago
Nice! How does squatting prevent the overriding Aorta from getting the deoxygenated blood? Does it increase the pressure in the left ventricle and force blood into the Pulmonary artery? Is that what you meant by afterload?
shalshah 1 year ago
Exactly! Increased afterload via crimping of the femoral arteries will increase the pressure against the LV ejection, and therefore push blood toward the area of less resistance (which upon squatting became the pulmonary artery). Before the squat, the path of least resistance was through the over-riding aorta.
JPShand 1 year ago