interesting ... might be helpful if you specified the kind of lipase you were talking about because of property differences.
For example, in acute pancreatitis, sometimes TGs skyrocket ( > several thousand ). In these patients, lipasemia occurs (e.g. >10,000 lipase level in the blood). Because pancreatic lipase degrades TGs, the question becomes why in these patients TGs are so high. The answer is that pancreatic lipase has an isoelectric point of 4.9-5.0, so it doesn't function.
@majik1213 Thank you for the comment. When I first researched about this mechanism, I was unable to find a mention of the specific lipase. It may have been known then or perhaps now, but the most I could derive was "some type of lipase". Great example by the way.
interesting ... might be helpful if you specified the kind of lipase you were talking about because of property differences.
For example, in acute pancreatitis, sometimes TGs skyrocket ( > several thousand ). In these patients, lipasemia occurs (e.g. >10,000 lipase level in the blood). Because pancreatic lipase degrades TGs, the question becomes why in these patients TGs are so high. The answer is that pancreatic lipase has an isoelectric point of 4.9-5.0, so it doesn't function.
majik1213 1 year ago
@majik1213 Thank you for the comment. When I first researched about this mechanism, I was unable to find a mention of the specific lipase. It may have been known then or perhaps now, but the most I could derive was "some type of lipase". Great example by the way.
illouie 1 year ago